Fig. 2From: Myelin repair in Alzheimer’s disease: a review of biological pathways and potential therapeuticsSimplified rendering of tau-associated pathways that may influence OL differentiation, including the binding of tau and Fyn to the OL cytoskeleton, which may simultaneously encourage hyperphosphorylation of tau, an upstream activator of Fyn, ITGB1, and other axonal and neurofilament proteins that may interact with tau to also promote OL differentiation beyond FynBack to article page