Fig. 7From: Suppression of astrocytic autophagy by αB-crystallin contributes to α-synuclein inclusion formationModel for the role of CRYAB in the clearance of protein aggregates in astrocytes via interaction with BAG3, HSC70 and HSPB8 under both physiological and pathological conditions. During aging and aging-related disease, deregulation of CRYAB in astrocytes may result in a suppression of autophagy leading to the accumulation of α-synuclein proteins and/or organellesBack to article page