Fig. 2

Mechanisms underlying the GCase–α-synuclein cross-talk that results in pathological conditions. GCase alterations induce disruptions of lipid composition and protein trafficking, inducing aggregation of α-synuclein
Mechanisms underlying the GCase–α-synuclein cross-talk that results in pathological conditions. GCase alterations induce disruptions of lipid composition and protein trafficking, inducing aggregation of α-synuclein